RightAnswer Knowledge Solutions Search Results for 79-06-1

New Search  |  Search Results (79-06-1)  |  Index of Example Chemical Results Pages
register now
RightAnswer Knowledge Solutions provides access to hundreds of data sources. Our premier and proprietary sources include fully-researched documents from well-established experts in the chemical and HazMat fields.

A search in our system for this chemical would return results – all in one place -- in the following categories from the listed data sources.
  • Chemical Identification
  • Environmental Hazards
  • First Aid/Medical Treatment
  • Handling/Storage/Shipping/Waste Management
  • MSDS Documents
  • Personal Protection
  • Physical Hazards/Corrective Response Actions
  • Physical/Chemical Properties
  • Regulatory/Standards/Labels
  • Report Abstracts and Studies
  • Reproductive Risk
  • Toxicology/Health Hazards/Exposure
Example of Acute Exposure data from MEDITEXT.

RightAnswer Proprietary Data Sources:

HAZARDTEXT™ Documentshelp
MEDITEXT® Documentshelp
REPROTEXT® Documentshelp


All Other Data Sources:

CCRIS Documentshelp
CHRIS Documentshelp
DART Documentshelp
• Acrylamide induced dominant lethality in mice following low dose chronic administration in drinking water.
• Acrylamide is not a selective developmental neurotoxicant in Sprague-Dawley rats.
• An evaluation of a number of agents affecting the nervous system.
Show More
More...
ECOTOX Documentshelp
ERG2016 Guidebookhelp
Fisher MSDShelp
• (4-Piperidinophenyl)methylamine
• 1,4-Bis(chloromethyl)-2,5-dimethoxybenzene
• 1-(3-Furyl)methanamine hydrochloride
Show More
More...
GENE-TOX Documentshelp
HSDB® Data Bankhelp
IRIS Documentshelp
LOLI® Listingshelp
NIOSH Documentshelp
New Jersey Fact Sheetshelp
NTP - Developmental Study Abstractshelp
NTP - Long-Term Studieshelp
NTP - Reproductive Toxicology Study Abstractshelp
OHM/TADS Documentshelp
REPROTOX® Documentshelp
RTECS® Registryhelp
Shepard's Cataloghelp
MSDSonline®help

ChemID External Links:

ATSDR Tox Portalhelp
Acrylamide
CAMEOhelp
Acrylamide
ChEBIhelp
Acrylamide
ClinicalTrials.govhelp
Acrylamide
CPDBhelp
Acrylamide
CTDhelp
Acrylamide
DARThelp
Acrylamide
DrugPortalhelp
Acrylamide
ECHAhelp
Acrylamide
eChemPortalhelp
Acrylamide
EMIChelp
Acrylamide
EPA ACToRhelp
Acrylamide
EPA Envirofactshelp
Acrylamide
EPA HPVIShelp
Acrylamide
EPA SRShelp
Acrylamide
ITERhelp
Acrylamide
MedlinePlusAllhelp
Acrylamide
MeSHhelp
Acrylamide
MeSH Headinghelp
Acrylamide
NIAID ChemDBhelp
Acrylamide
NIOSH ICSChelp
Acrylamide
NIST WebBookhelp
Acrylamide
OSHA Chemhelp
Acrylamide
OSHA ChemDBhelp
Acrylamide
PubChemhelp
Acrylamide
PubMedhelp
Acrylamide
PubMed AIDShelp
Acrylamide
PubMed Cancerhelp
Acrylamide
PubMed Toxicologyhelp
Acrylamide
SRC BIODEGhelp
Acrylamide
SRC CHEMFATEhelp
Acrylamide
SRC DATALOGhelp
Acrylamide
TOXLINEhelp
Acrylamide
TOXMAPhelp
Acrylamide
TRI2000help
Acrylamide
TRI2001help
Acrylamide
TRI2002help
Acrylamide
TRI2003help
Acrylamide
TRI2004help
Acrylamide
TRI2005help
Acrylamide
TRI2006help
Acrylamide
TRI2007help
Acrylamide
TRI2008help
Acrylamide
TRI2009help
Acrylamide
TRI2010help
Acrylamide
TRI2011help
Acrylamide
TRI2012help
Acrylamide
TRI2013help
Acrylamide
TRI95help
Acrylamide
TRI96help
Acrylamide
TRI97help
Acrylamide
TRI98help
Acrylamide
TRI99help
Acrylamide
USA.govhelp
Acrylamide
WebWISERhelp
Acrylamide

Other Government Links Searched via RegsKnowledge:

State Environmental Regulationshelp
CFR Regulationshelp

Example Content from MEDITEXT for 79-06-1:


Please note: this is an extract of information from a larger document. Full document and details are available by subscription.

ACUTE EXPOSURE INFORMATION

  1. WITH POISONING/EXPOSURE
    1. Toxic effects depend on the duration, total dose and rate of exposure. The effects of acute high-dose exposure may be delayed in onset for several hours. Following large exposures, these include somnolence, confusion, hallucinations, disorientation, incoordination, tremors, and possibly seizures with cardiovascular collapse. Peripheral neuropathy may appear several weeks following significant acute exposure or following significant chronic exposures. Encephalopathy may occur in severe acute poisonings.
    1. In sub-acute toxicity (exposure over days to weeks), and if of sufficient concentration, drowsiness, somnolence, loss of concentration, truncal ataxia, dysarthria, nystagmus, and urinary retention may occur. Polyneuropathy and peripheral neuropathy, with mainly motor and proprioceptive disturbances, may follow several weeks later.
    1. Neurotoxic effects may include muscle weakness, numbness of limbs and extremities, tingling fingers, speech difficulties, unsteadiness, tremors, fatigue, lethargy, memory difficulties, and a sensory polyneuropathy if of sufficient dose. Excessive sweating is also common after exposure.
    1. Dermal contact is a common route of exposure and may result in skin irritation with numbness, tingling, blistering and peeling with direct contact of high concentrations. Visual impairment and eye irritation also occur with significant exposure. Inhalation may produce a cough and sore throat. Ingestion, the least common route, may result in abdominal pain.
    1. Complete recovery over a few weeks to months may be expected with mild symptoms, including prolonged weakness, but in cases of severe exposure, gradual and incomplete recovery may occur with residual ataxia, loss of reflexes, distal extremity weakness, and sensory disturbances.
© 2011-2017 RightAnswer.com, Inc. and/or its licensors. All rights reserved. No claim to original U.S. Govt. works.