RightAnswer Knowledge Solutions provides access to hundreds of data sources. Our premier and proprietary sources include fully-researched documents from well-established experts in the chemical and HazMat fields.
A search in our system for this chemical would return results – all in one place -- in the following categories from the listed data sources.
- Chemical Identification
- Environmental Hazards
- First Aid/Medical Treatment
- Handling/Storage/Shipping/Waste Management
- Personal Protection
- Physical Hazards/Corrective Response Actions
- Physical/Chemical Properties
- Report Abstracts and Studies
- Reproductive Risk
- Toxicology/Health Hazards/Exposure
|Example of Acute Exposure data from MEDITEXT.|
Other Government Links Searched via RegsKnowledge:
State Environmental Regulations
Example Content from MEDITEXT for 630-08-0:
Please note: this is an extract of information from a larger document. Full document and details are available by subscription.
ACUTE EXPOSURE INFORMATION
- SOURCES: Odorless, colorless gas produced by the incomplete combustion of any carbon containing substance. Common sources include household fires, home furnaces, stoves and water heaters, and vehicle exhaust. Another potential source is methylene chloride (often used as a paint stripper or degreaser) that is absorbed through inhalation, ingestion, or dermal contact and is subsequently metabolized by the liver to carbon monoxide.
- PHARMACOLOGY: Binds to hemoglobin with an affinity approximately 250 times greater than that of oxygen.
- TOXICOLOGY: Impairs oxygen delivery, producing cellular hypoxia and ischemia.
- EPIDEMIOLOGY: Common poisoning; one of the leading toxicologic causes of death.
- WITH POISONING/EXPOSURE
- MILD TO MODERATE TOXICITY: Headache, nausea, dizziness, vomiting, weakness, and confusion are often reported, with headache being the most common.
- SEVERE TOXICITY: Coma, syncope, seizures, cardiac dysrhythmias, myocardial ischemia, and death result from more severe poisonings and reflect damage to the organ systems (brain and heart) with the highest oxygen demand. Delayed neurocognitive effects, which include dementia, amnestic syndromes, psychosis, parkinsonism, chorea, apraxia, neuropathies, difficulty concentrating, and personality changes, can occur from 2 to 40 days following the initial exposure. Unfortunately, there are no good predictive markers for who will develop neurocognitive sequelae, including the initial carboxyhemoglobin level or the severity of the initial poisoning.
© 2011-2017 RightAnswer.com, Inc. and/or its licensors. All rights reserved. No claim to original U.S. Govt. works.